All orders can be given here, at which point you will be contacted by ICAD to ascertain the year and colour of the award, what it was received for and any other pertinent details. Twenty-four were male (67) and the mean age was 63.5☑3.7 years. Preoperative metrics and outcome are presented in tables 1 and 2. Replica bells are available to ICAD award winners, they will be subtly engraved to indicate that they are a replica. Between August 2008 and August 2015, 435 patients received thrombectomy, of whom 36 (8.3) were found to have underlying ICAD during the thrombectomy procedure. ICAD bells have been cast in the same bronze foundry in Dublin since the 1980s. The ICAD logo appears on the front of the bell and the year of the award on the back. Gold, Silver, Bronze, Green (the Greenhorn award) and Blue (the Client awards) are each treated differently to achieve their finish. The original ICAD bell, designed by Jarlath Hayes, was made of beaten metal. Intracranial vasculopathy magnetic resonance imaging vascular diseases.For nearly sixty years, the ICAD Bell has remained iconic – a prestigious and coveted prize and acknowledged internationally as a benchmark of creative excellence. Multicontrast VWI can be a complementary tool for intracranial vasculopathy differentiation, which often leads to more invasive workups when reversible cerebral vasoconstriction syndrome and vasculitis are in the differential diagnosis. Combining T1 and T2 VWI increased the sensitivity of VWI in differentiating ICAD from other vasculopathies from 90.1% to 96.3%. There were also significant differences in the presence, intensity, and pattern of enhancement between all lesion types. ICAD lesions were significantly more likely to have eccentric wall involvement (90.1%) than reversible cerebral vasoconstriction syndrome (8.2% P<0.001) and vasculitic lesions (6.7% P<0.001) and were also more likely to have T2 hyperintensity present than the other 2 vasculopathies (79% versus 0% P<0.001). There was substantial to excellent inter-reader agreement for the assessment of lesional T2 hyperintensity (κ=0.80), pattern of wall thickening (κ=0.87), presence (κ=0.90), pattern (κ=0.73), and intensity (κ=0.77) of enhancement. Twenty-one cases of atherosclerosis (intracranial atherosclerotic disease ), 4 of reversible cerebral vasoconstriction syndrome, and 4 of vasculitis were identified, with a total of 118 stenotic lesions (81 ICAD, 22 reversible cerebral vasoconstriction syndrome, and 15 vasculitic lesions). Two blinded experts evaluated T1 precontrast and postcontrast and T2-weighted VWI characteristics, including the pattern of wall thickening presence, pattern, and intensity of postcontrast enhancement and T2 signal characteristics. We retrospectively reviewed patients with clinically defined intracranial vasculopathies causing luminal stenosis/irregularity who underwent VWI studies. This study uses T1- and T2-weighted VWI to differentiate intracranial vasculopathies. Although studies have attempted to differentiate intracranial vascular disease using vessel wall magnetic resonance imaging (VWI), none have incorporated multicontrast imaging.